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This imbalance might cause LPS to be increasingly produced and enter the bloodstream, triggering widespread inflammation through IL-10, disruptions in the HPA axis, and a reduction in cortisol levels, another factor linked with ME/CFS [79, 80]

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73 ESR1 and TP53 driver mutations, as well as amplifications in MDM4 , FGFR1 and CCND1 , were detected in patients relapsing after endocrine therapy, and mutations in SWI/SNF genes were found following taxane chemotherapy